PRESS RELEASE
Several readers of CluelessDoctors asked me to write about statins. I wrote a complete essay on the subject at here that explains all you ever wanted and deeded to know about statins.
Is there anyone who doesn’t need statins?
Must we take statins? Some researchers and doctors most certainly think so. Some even recommend adding statins to drinking water. This is obviously a rather dangerous proposition. After all, if we really don’t need cholesterol, why does our body make it? And if statins are so essential that they should be in the water to drink by all people in all life stages, then how come that our body didn’t add statin as an essential nutrient as part of evolution?
Can we suffer from statin deficiency?
Have you heard of NNT (number needed to treat) and NNH (number needed to harm)? I examine these in the context of statins in my article. I also list some adverse effects—some very serious. Did you know that statins have 300 known adverse events?
I also discuss a big-pharma trick that is not so funny. CoQ10 production is blocked by statins (as is the production of steroids, vitamin D, and other important hormones). CoQ10 is heart protective so one would think that statins, which are supposed to protect the heart, would support CoQ10. Right? Wrong. Statins block the production of CoQ10, because it is a step in the chain of steps that generates cholesterol. Not having enough CoQ10 also harms mitochondria; taking statins prevents proper energy production–this explains the muscle damage statins cause.
One big-pharma considered adding CoQ10 to their statin formulation. Read what happened next.
Also discuss are what heart disease and cholesterol are and how—if at all—they connect in a meaningful way; what high LDL is and if that is really a bad thing. I explain what atherosclerosis is. While you may think you know what it is, you may find some surprising information in what I wrote.
Let me know what you think about the article and ask questions here or there. Receiving feedback on what you want me to write about is super! Keep on having your requests coming! 😊
Comments are welcome, as always, and are moderated for appropriateness
Angela
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About Angela A Stanton, Ph.D.
Angela A Stanton, PhD, is a Neuroeconomist focusing on chronic pain--migraine in particular--physiology, electrolyte homeostasis, nutrition, and genetics. She lives in Southern California. Her current research is focused on migraine cause, prevention, and treatment without the use of medicine. As a forever migraineur from childhood, her discovery was helped by experimenting on herself. She found the cause of migraine to be at the ionic level, associated with disruption of the electrolyte homeostasis, resulting from genetic variations of all voltage dependent channels, gates, and pumps (chanelopathy) that modulate electrolyte mineral density and voltage in the brain. In addition, insulin and glucose transporters, and several other variants, such as MTHFR variants of B vitamin methylation process and many others are different in the case of a migraineur from the general population.
Migraineurs are glucose sensitive (carbohydrate intolerant) and should avoid eating carbs as much as possible. She is working on her hypothesis that migraine is a metabolic disease.
As a result of the success of the first edition of her book and her helping over 5000 migraineurs successfully prevent their migraines world wide, all ages and both genders, and all types of migraines, she published the 2nd (extended) edition of her migraine book "Fighting The Migraine Epidemic: Complete Guide: How To Treat & Prevent Migraines Without Medications". The 2nd edition is the “holy grail” of migraine cause, development, and prevention, incorporating all there is to know. It includes a long section for medical and research professionals. The book is full of academic citations (over 800) to authenticate the statements she makes to make it easy to follow up by those interested and to spark further research interest. It is a "Complete Guide", published on September 29, 2017.
Dr. Stanton received her BSc at UCLA in Mathematics, MBA at UCR, MS in Management Science and Engineering at Stanford University, PhD in Economics with dissertation in neuroscience (culminating in Neuroeconomics) at Claremont Graduate University, fMRI certification at Harvard University Medical School at the Martinos Center for Neuroimaging for experimenting with neurotransmitters on human volunteers, certification in LCHF/ketogenic diet from NN (Nutrition Network), certification in physiology (UPEN via Coursea), Nutrition (Harvard Shool of Public Health) and functional medicine studies. Dr. Stanton is an avid sports fan, currently power weight lifting and kickboxing. For relaxation (yeah.. about a half minute each day), she paints and photographs and loves to spend time with her family of husband of 45 years, 2 sons and their wives, and 2 granddaughters.
Follow her on Twitter at: @MigraineBook, LinkedIn at https://www.linkedin.com/in/angelaastantonphd/ and facebook at https://www.facebook.com/DrAngelaAStanton/
Clueless Doctors & Scientists 
I was lucky in that I never got any side effects – I stopped and restarted them a couple of times to check – all they did was reduce my LDL.
I stopped them for good when I realised how they achieve this – by shutting down the entire mevalonate pathway.
Here’s entertaining – when I went hyperthyroid my LDL dropped by exactly as much as the statin, so maybe they should put thyroxine in the drinking water. Er. I think not. An elderly (retired) doctor pointed out that back in the day when they saw high “cholesterol” they would assume hypothyroid and do a TSH. Then when low fat diets and metabolic syndrome were invented this connection went away. Strangely there is cardiovascular risk with both high and low thyroid.
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Indeed, both low and high cholesterol are bad for good reasons: we must have cholesterol and a healthy body produces as much as we need. 🙂 If we produce more: we are not healthy. Ditto if we produce less.
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Yes and when the lipoproteins are damaged they don’t work properly (miss the receptors) so the body will generate more in hopes that enough undamaged ones get through. Stop the damage – oxidation and glycation among others – and things revert to working properly. Then oxidation and glycation are among the things that damage other tissues which the body attempts to repair with LDL. When the LDL is also damaged there is a runaway feedforward loop. Statins affect the wrong part of the circuit.
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exactly!
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