Are You Dumifying Yourself Taking Statins?

You bet!

Dumifying it is!

The Scientific American released an awesome little capture that by now everyone should know with the title: Do Statins Produce Neurological Effects?  Since you may not have subscription to this magazine, let me summarize the findings by quoting a few things into one sentence:

“Statins… produce neurological effects… studies show that statins can influence our sleep and behavior—and perhaps even change the course of neurodegenerative conditions, including dementia. The most common adverse effects include muscle symptoms, fatigue and cognitive problems… peripheral neuropathy—burning, numbness or tingling in their extremities—poor sleep, and greater irritability and aggression… statins generally reduce the risk of ischemic strokes… but can also increase the risk of hemorrhagic strokes, or bleeding into the brain…[they] also appear to increase or decrease aggression [based on gender]… women taking statins… showed increased aggression; men… less, possibly because of reduced testosterone levels… Statins may also affect neurodegenerative disorders, such as dementia, Parkinson’s disease or amyotrophic lateral sclerosis (ALS)… some patients taking statins develop ALS or ALS-like conditions with progressive muscle wasting… statins cause increases or decreases in tissue damage known as oxidative stress, involved in neurodegenerative diseases.”

Do you have further questions? If yes, ask away!

As far as I am concerned, statins should be trashed by all, except the few with dangers for ischemic stroke (blood clot). Everyone else just gets dummyfied so stop taking the thing! Stay smart!

Comments welcome as always,



About Angela A Stanton, Ph.D.

Angela A Stanton, PhD, is a Neuroeconomist focusing on chronic pain--migraine in particular--physiology, electrolyte homeostasis, nutrition, and genetics. She lives in Southern California. Her current research is focused on migraine cause, prevention, and treatment without the use of medicine. As a forever migraineur from childhood, her discovery was helped by experimenting on herself. She found the cause of migraine to be at the ionic level, associated with disruption of the electrolyte homeostasis, resulting from genetic variations of all voltage dependent channels, gates, and pumps (chanelopathy) that modulate electrolyte mineral density and voltage in the brain. In addition, insulin and glucose transporters, and several other variants, such as MTHFR variants of B vitamin methylation process and many others are different in the case of a migraineur from the general population. Migraineurs are glucose sensitive (carbohydrate intolerant) and should avoid eating carbs as much as possible. She is working on her hypothesis that migraine is a metabolic disease. As a result of the success of the first edition of her book and her helping over 5000 migraineurs successfully prevent their migraines world wide, all ages and both genders, and all types of migraines, she published the 2nd (extended) edition of her migraine book "Fighting The Migraine Epidemic: Complete Guide: How To Treat & Prevent Migraines Without Medications". The 2nd edition is the “holy grail” of migraine cause, development, and prevention, incorporating all there is to know. It includes a long section for medical and research professionals. The book is full of academic citations (over 800) to authenticate the statements she makes to make it easy to follow up by those interested and to spark further research interest. It is a "Complete Guide", published on September 29, 2017. Dr. Stanton received her BSc at UCLA in Mathematics, MBA at UCR, MS in Management Science and Engineering at Stanford University, PhD in Economics with dissertation in neuroscience (culminating in Neuroeconomics) at Claremont Graduate University, fMRI certification at Harvard University Medical School at the Martinos Center for Neuroimaging for experimenting with neurotransmitters on human volunteers, certification in LCHF/ketogenic diet from NN (Nutrition Network), certification in physiology (UPEN via Coursea), Nutrition (Harvard Shool of Public Health) and functional medicine studies. Dr. Stanton is an avid sports fan, currently power weight lifting and kickboxing. For relaxation (yeah.. about a half minute each day), she paints and photographs and loves to spend time with her family of husband of 45 years, 2 sons and their wives, and 2 granddaughters. Follow her on Twitter at: @MigraineBook, LinkedIn at and facebook at
This entry was posted in Big Pharma, Drugs of Shame, Healthcare, Interesting reading, Must Read, Thoughts and tagged , , , , , , , , . Bookmark the permalink.

15 Responses to Are You Dumifying Yourself Taking Statins?

  1. Christopher says:


    You say: “As far as I am concerned, statins should be trashed by all, except the few with dangers for ischemic stroke (blood clot)”

    Many many strokes are caused by blood clots but surely for that blood thinners (or lifestye changes etc) are the ‘remedy’ not statins? What’s the (supposed) link between statins and blood clots?

    I know the article is very old so maybe you won’t reply….

    Liked by 1 person

    • Oh I try to answer all comments Christopher. 🙂

      I agree with you. This was written at the time when I didn’t yet understand the full picture well enough to explain it well enough. I suppose what I should have written there is a bit of better definition of what I meant by blood clots.

      People who take blood thinners need to reduced their clotting ability by preventing platelet formation–so those drugs interfere with platelet creation. That condition has nothing to do with statins, I completely agree. The thing I was referring to here as “clots” are not made from clotted blood as much as what breaks off from a calcified plaque in the arteries in an atherosclerotic artery. That “clot”, which is called a clot just like a blood clot, is not made from platelets but from a mix of calcium, platelets, cholesterol, as well as macrophages and other goodies.

      It seems that statins have a stabilizing effect over these types of “clots”.

      I hope this makes sense.



      • Chris says:

        Ok… ( thank you for your quick response) Having searched your blog I have a general question…

        Is it your view that statins have ANY use at all ( except very mild anti inflammatory)?

        If so ( having a use) to what age, gender and health person ( eg secondary prevention)?

        I have posted details of your blog in the Facebook group Stopped our Statins and praised how quickly you respond. ( I thought I should mention that before you answered! )

        Perhaps even a major blog post on statins is needed / here’s a prompt:

        Liked by 1 person

        • Thanks for the link to that post Chris. That is one article that should never see daylight. Silly. But let me start with the beginning of your questions.

          Statins have a very limited use for a very rare condition that is called familial hyperlipidemia, something that has completely been taken out of context in recent years. Originally this condition refers to extremely high LDL cholesterol (over 400–US measure) and has some side effects: chest pain with activity, xanthomas (fatty deposits often found in many places, including around the eyelids) and gray-white cholesterol deposits around the corneas (this is something, by the way, I also have but only this and nothing else.

          For people with this condition, cholesterol, including triglycerides, which are fats and not cholesterol, can go so high that a blood sample may look lighter than red and may be thicker… sort of milky in extreme cases. This is a very rare condition but in cases like this, the body has a genetic control mechanism not working properly and so more cholesterol is made than needed and the body lacks the ability to clear cholesterol. In this case, reducign cholesterol production makes a lot of sense, hence statins can be useful. However, this condition is pretty self limiting–if untreated–with early heart attacks and similar–before statins were available and before proper diagnosis was available.

          Today, we have a problem on many levels when it comes to cholesterol.

          1) When we test cholesterol, we are testing the wrong thing. LDL, HDL, and triglycerides… where is the cholesterol??? LDL = Low Density Lipoprotein (it is a protein), HDL = High density lipoprotein (it is a protein), and triglycerides are… well 3 fatty acids connected with a glycerol head. So where is the cholesterol?

          2) Assume that all doctors know that none of what a cholesterol blood tests measure is cholesterol (they don’t but assume for a second that they do): what does the cholesterol test tell them?

          3) Assume that the cholesterol test actually does measures cholesterol: if we believe that HDL is good and LDL is bad and we know that triglycerides are fats and not cholesterol, what does the sum of all (referred to as total cholesterol) tell us? Good + Bad + Fat = All Bad? Why? Does the sum of good and bad cholesterol make any sense based on which we can make a decision if someone needs statins? If someone has very high good HDL, and therefore the total cholesterol is high: your doctor will want to put you on statins, which will then lower your good cholesterol and make you sick in the process. See what I mean?

          4) Why do we need to have low LDL? Do clinical trials show that those with lower LDL gain any benefits? Actually the opposite is true–see the image here: and look under the bell curve for LDL. I cannot attach an image here so I will write an article on this later–I changed that image where the link goes with stuff written on it for clarity. To answer my question: there have never been any clinical trials to prove that higher LDL causes (or is even associated with) a higher rate of heart attacks. There have only been data analyses of cholesterol and heart attack patients, with whom they never checked what else may be the matter. Kraft found that all heart attack victims have T2D that is undiagnosed. May it be T2D that is the cause of those heart attacks? If so, taking statins is a shot in the foot because it causes T2D!!

          I will definitely need to write a blog on this, since I can see my comment is moving toward becoming an article in itself. 😉 One day.

          Thanks for sharing my blog in the FB group for stopping statins.

          As for the 75-year olds and over: there is a golden rule. never ever give a person over 70 statins. At that age–no matter what origin–cholesterol has a protective effect. And lastly: never ever give a woman statins. Cholesterol is protective to all women. Ironically–I must add–our brain’s white matter is all cholesterol and is the most important part of our brain.


      • chris says:

        I thought I’d posted a follow up – a suggestion to have a major article on statins – especially in view of today’s media blitz on statins for the over 75s…

        Liked by 1 person

        • I think it was part of the original comment you made–but I had no time yet to read all the comments. Today has been an extremely busy day and i still have comments to approve and respond to. 🙂

          I will definitely write a blog on statins. I can see it is really needed.


  2. Roald Michel says:

    Yeah, fuck them Statins. 😳

    Btw, one of the experts crossing words with me, me a.k.a. The Humble One, said that Strattera and Concerta are SSRIs. I have my doubts about that ❓ and 😕 So tell me, is he right?

    Liked by 1 person

    • Be Healthy says:

      Stratera is an SNRI (serotonin & norepinephrine reuptake inhibitor) and Concerta is Ritalin, which is a NDRI (norepinephrine & dopamine reuptake Inhibitor) so neither is an SSRI but similar is actions only not ONLY on serotonin. Ritalin also has some other tricks–it is a really bad drug. Both are bad. So he is wrong since neither is and SSRI but right in that both inhibit reuptake and thus flood the brain with stuff, Very strong “discontinuation syndrome” (love this political correctness BS). Sure sounds nicer than addiction!!! 👿

      Agree with your statement on statins…


I would love to see your thoughts!

Fill in your details below or click an icon to log in: Logo

You are commenting using your account. Log Out /  Change )

Twitter picture

You are commenting using your Twitter account. Log Out /  Change )

Facebook photo

You are commenting using your Facebook account. Log Out /  Change )

Connecting to %s

This site uses Akismet to reduce spam. Learn how your comment data is processed.